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2015). These experiments, taken alongside one another, propose that, although a direct activation of GPR55 or PPAR‐α occurs, PEA can generate its anti‐inflammatory action within the gut also by using oblique activation of CB1 and CB2 receptors, likely resulting from the power of this compound to potentiate the motion of endocannabinoids at the
The next pain stage is due to an inflammatory reaction, though the key response is acute harm on the nerve fibers. Conolidine injection was found to suppress equally the period 1 and a couple of pain response (60). This means conolidine proficiently suppresses both of those chemically or inflammatory soreness of each an acute and persistent nature.